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AICAR, aminoimidazole carboxamide ribonucleotide, acts as an agonist to AMP-activated protein kinase; AMP-activated protein kinase, also known as AMPK, is an enzyme with an important role in cellular homeostasis and energy regulation.[1] AMPK acts through a variety of means to ultimately stimulate liver fatty oxidation, ketogenesis, beta-cell modulation of insulin secretion, and other functions within the body. AICAR has been shown to stimulate glucose uptake and reduce apoptosis by reducing reactive oxygen compounds within cells.

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AICA ribonucleotide or AICAR (aminoimidazolecarboxamideribonucleotide) is an intermediate in the generation of inosine monophosphate, which acts as an AMP-activated protein kinase agonist.[1] It stimulates glucose uptake and increases the activity of p38 mitogen-activated protein kinases ? and ? in skeletal muscle tissue,[2] as well as suppressing apoptosis by reducing production of reactive oxygen compounds inside the cell.[3] In 2008, researchers at the Salk Institute discovered that AICAR given to experimental mice significantly improves their performance in endurance-type exercise, apparently by converting fast-twitch muscle fibers to the more energy-efficient, fat-burning, slow-twitch type. They also looked at the administration of GW 501516 (also called GW1516) in combination with AICAR. Given to mice that did not exercise, this combination activated 40% of the genes that were turned on when mice were given GW1516 and made to exercise. This suggests it may be possible to obtain some of the benefits of exercising without actually exercising.[4] Because of the enhanced endurance effects, this could potentially be used by athletes to enhance their performance.

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AICAR, Aminoimidazol carboxamid Ribonukleotid, wirkt als ein Agonist auf AMP-aktivierte Proteinkinase; AMP-aktivierte Proteinkinase, die auch als AMPK bekannt ist, ist ein Enzym, das eine wichtige Rolle in der zellulären Homöostase und Energieregelung. [1] AMPK wirkt durch eine Vielzahl von Mitteln, um letztlich zu stimulieren Leberfettoxidation Ketogenese, beta-Zellen-Modulation der Insulin Sekretion und andere Funktionen innerhalb des Körpers. AICAR wurde gezeigt, dass die Glukoseaufnahme zu stimulieren und Apoptose durch Reduzierung reaktiver Sauerstoffverbindungen innerhalb der Zellen.




Nach wissenschaftlichen Studie, die von Tierversuchspersonen basiert worden ist, wird Aicar Funktionalität zu verhindern, dass die Produktion der Enzyme Adenosinkinase (ADK) und Adenosindeaminase (ADA) verbunden. Dieser Prozess letztendlich wirkt, um die Glukoseaufnahme zu stimulieren und die die Aktivität von p38 Mitogen-aktivierte Proteinkinasen a und β in Skelett- und Muskel Gewebe. Auf dem Weg hat es sich gezeigt, dass Apoptose durch Senkung der Produktion von Sauerstoffverbindungen, die innerhalb der Zelle angeordnet werden, zu blockieren. Diese Prozesse liefen letztlich auf der Durchblutung Verordnung, die wiederum ermöglicht eine Tierversuchsperson, eine verbesserte Geschwindigkeit der Homöostase erleben zu steigern.






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AICA Ribonukleotid oder AICAR (aminoimidazolecarboxamideribonucleotide) ist ein Zwischenprodukt bei der Erzeugung von Inosinmonophosphat, die als AMP-aktivierte Proteinkinase-Agonist wirkt. [1] Es stimuliert die Glukoseaufnahme und erhöht die Aktivität von p38-Mitogen-aktivierte Proteinkinasen? und? in Skelettmuskelgewebe, [2] sowie die Unterdrückung von Apoptose durch Reduzierung Produktion von reaktiven Sauerstoffverbindungen in der Zelle. [3] Im Jahr 2008 Forscher am Salk Institute entdeckte, dass AICAR experimentelle Mäuse, denen ihre Leistung im Ausdauertyp erheblich verbessert Bewegung, offenbar durch die Umwandlung schnell zuckenden Muskelfasern an den energieeffizienter, Fettverbrennung, langsam kontrahierenden Typs. Sie betrachteten auch die Verwaltung von GW 501516 (auch GW1516) in Kombination mit AICAR. Gegeben, um Mäuse, die nicht ausüben, aktiviert diese Kombination 40% der Gene, die auf, wenn Mäuse erhielten GW1516 gedreht wurden und machte zu trainieren. Dies legt nahe, kann es möglich sein, einige der Vorteile der Wahrnehmung ohne tatsächlich Gebrauch zu erhalten. [4] Wegen der verbesserten Dauereffekte, könnte dies potentiell von Sportlern verwendet werden, um ihre Leistung zu verbessern.

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According to scientific study that has been based on animal test subjects, AICA Ribonucleotide’s functionality is linked to preventing the production of the enzymes Adenosine kinase (ADK) and Adenosine deaminase (ADA).  This process ultimately acts to stimulate glucose uptake and boost the activity of p38 mitogen-activated protein kinases α and β in skeletal and muscular tissue.  Along the way, it has been shown to block apoptosis by lowering the production of oxygen compounds that are located within the cell.  These processes ultimately lead to boost in blood flow regulation, which in turn enables an animal test subject to experience an improved rate of homeostasis.






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